P681 mutations within the polybasic motif of spike dictate fusogenicity and syncytia formation of SARS CoV-2 variants
Taube Ran 1Korovin Dina 2Mandelboim Michal 3Atari Nofar 4Rosental Benyamin 1lass Ido Cohen 1Ottolenghi Aner 2Rosenberg Elli 5Kuzmina Alona1
作者信息
- 1. The Shraga Segal Department of Microbiology Immunology and Genetics Faculty of Health Sciences, Ben-Gurion University of the Negev
- 2. The Shraga Segal Department of Microbiology Immunology and Genetics Faculty of Health Sciences, Ben-Gurion University of the Negev||Regenerative Medicine and Stem Cell Research Center, Ben Gurion University of the Negev
- 3. Central Virology Laboratory, Public Health Services, Ministry of Health and Sheba Medical Center||Department of Epidemiology and Preventive Medicine, School of Public Health, Sackler Faculty of Medicine
- 4. Central Virology Laboratory, Public Health Services, Ministry of Health and Sheba Medical Center
- 5. Soroka Medical Center
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Key words
SARS CoV-2/Fusion and Syncytia/Pfizer-BTN162b2/neutralization antibodies/Furin-Cleavage Site/Poly basic domain引用本文复制引用
Taube Ran,Korovin Dina,Mandelboim Michal,Atari Nofar,Rosental Benyamin,lass Ido Cohen,Ottolenghi Aner,Rosenberg Elli,Kuzmina Alona.P681 mutations within the polybasic motif of spike dictate fusogenicity and syncytia formation of SARS CoV-2 variants[EB/OL].(2025-03-28)[2025-12-13].https://www.biorxiv.org/content/10.1101/2022.04.26.489630.学科分类
基础医学/分子生物学/微生物学
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